| ID | Sequence | Length | GC content |
|---|---|---|---|
| LINC01504:1 | CUUAGCCCUCCCAGAAACUUACUGGGUCAUUUUGACUGCAAGUGACAGA… | 1742 nt | 0.4650 |
| LINC01504:2 | GAAAGAGGAAAUGCUGCAGAGACCCAGCCGGAAACAGUGCUGGCCAGCU… | 672 nt | 0.4955 |
| LINC01504:3 | AAAGAGGAAAUGCUGCAGAGACCCAGCCGGAAACAGUGCUGGCCAGCUC… | 671 nt | 0.4948 |
| LINC01504:4 | GAAAUGCUGCAGAGACCCAGCCGGAAACAGUGCUGGCCAGCUCACGCCU… | 2078 nt | 0.4230 |
| LINC01504:5 | GAAAUGCUGCAGAGACCCAGCCGGAAACAGUGCUGGCCAGCUCACGCCU… | 673 nt | 0.4978 |
| LINC01504:6 | UCUGAAGAGCUAAAGACUUCCUGAAAUUUAAAAAUUAGUGCUUUUAUUU… | 588 nt | 0.4864 |
| LINC01504:7 | CAGAAACAACCACGGUUACCCUAGAAGUAAAUCCAGAGCUGCGGGACGC… | 666 nt | 0.4910 |
No relevant information is available at the moment.
A study in humans identified LINC01504 as a long non-coding RNA that is upregulated in myocardial fibrosis compared to acute myocardial infarction, suggesting a mechanistic role in this pathological progression [Wang et al. DOI:10.3389/fcvm.2021.664044].