| ID | Sequence | Length | GC content |
|---|---|---|---|
| LINC02397:30 | AGUUCCCCACAGCACAGAGAGGAAGCAAGGUCUUUGCAUGCCCUGUUCA… | 7622 nt | 0.4049 |
| LINC02397:31 | GCGUAAUUCAAGAGCCCUGACUCUUUCACCAUUUCAUGAUGGCUUAUUA… | 239 nt | 0.4979 |
| LINC02397:32 | GAGCCCUGACUCUUUCACCAUUUCAUGAUGGCUUAUUACUCGCACACGG… | 1453 nt | 0.4212 |
| LINC02397:33 | ACUCUUUCACCAUUUCAUGAUGGCUUAUUACUCGCACACGGCAUUGUCA… | 10260 nt | 0.3903 |
| LINC02397:34 | ACUCUUUCACCAUUUCAUGAUGGCUUAUUACUCGCACACGGCAUUGUCA… | 9922 nt | 0.3875 |
| LINC02397:6 | AUUUGAAUUGGUGAACUUAGUAAAGCAGACGGCUCUCACCAAUAAGGGC… | 2817 nt | 0.3848 |
| LINC02397:8 | GAAGGUAAAAAAAAGAGGGAGGUCUGAGAAAUAGAAAUAUCAGAGGAAG… | 3897 nt | 0.3711 |
No relevant information is available at the moment.
A study in humans demonstrated that the LINC02397 was down-regulated in peripheral blood mononuclear cells from patients with ST-elevation myocardial infarction compared to those with non-ST-elevation myocardial infarction, with a fold change of 2.35, and was identified as a diagnostic biomarker for acute myocardial infarction [Zhong et al. DOI:10.1097/MD.0000000000013066].