| ID | Sequence | Length | GC content |
|---|---|---|---|
| lnc-RNASEH1-1:10 | CAGGAAGGAGCCUCUGCAGAAACGGAACCAGCGGGAACCUUGACCUGAG… | 914 nt | 0.5361 |
| lnc-RNASEH1-1:11 | GGACCUGAGAGUGGGCAGCCACUACCUGAAGCUCUUGGAACAGAACCAG… | 436 nt | 0.5000 |
| lnc-RNASEH1-1:12 | UGUGAAGAAUGCUGCUGUGAACAGGGGAGUGCCCCUGCCUGUUUGGGUC… | 482 nt | 0.4979 |
| lnc-RNASEH1-1:13 | CAGCCAGUGCUGCAGGUCAGGGGCCGCCAUCCCCCAGAGCUGCUGUGAG… | 584 nt | 0.5000 |
| lnc-RNASEH1-1:7 | CUGACCCGAUGGGAUGGGUGUCCCUAUAAGGAGAGACCCCAGAGUGCUC… | 423 nt | 0.5532 |
| lnc-RNASEH1-1:8 | GAGCCAGGAAGGAGCCUCUGCAGAAACGGAACCAGCGGGAACCUUGACC… | 789 nt | 0.4728 |
| lnc-RNASEH1-1:9 | GAGCCAGGAAGGAGCCUCUGCAGAAACGGAACCAGCGGGAACCUUGACC… | 791 nt | 0.4716 |
No relevant information is available at the moment.
A study in humans identified the lnc-RNASEH1-1 as a long non-coding RNA that was down-regulated in peripheral blood mononuclear cells of patients with ST-elevation myocardial infarction compared to those with non-ST-elevation myocardial infarction, with a fold change of 16.41 [Zhong et al. DOI:10.1097/MD.0000000000013066].