| ID | Sequence | Length | GC content |
|---|---|---|---|
| LINC01270:1 | AUUAAUGACCCGGAGCCACAUGACGGAGCAUGACACAGCACUUUUGGGC… | 2281 nt | 0.5011 |
| LINC01270:10 | AUUAAUGACCCGGAGCCACAUGACGGAGCAUGACACAGCACUUUUGGGC… | 2281 nt | 0.5011 |
| LINC01270:11 | AUUAAUGACCCGGAGCCACAUGACGGAGCAUGACACAGCACUUUUGGGC… | 5002 nt | 0.4824 |
| LINC01270:12 | AUUAAUGACCCGGAGCCACAUGACGGAGCAUGACACAGCACUUUUGGGC… | 8912 nt | 0.4900 |
| LINC01270:13 | AGACGGCCAUCCCCCAGGCCCAGCCUUGCCCACAAGAUUCCCAGAUUUC… | 10599 nt | 0.4974 |
| LINC01270:14 | AGACGGCCAUCCCCCAGGCCCAGCCUUGCCCACAAGAUUCCCAGAUUUC… | 9327 nt | 0.4934 |
| LINC01270:15 | AGACGGCCAUCCCCCAGGCCCAGCCUUGCCCACAAGAUUCCCAGAUUUC… | 11704 nt | 0.5067 |
| LINC01270:16 | AGACGGCCAUCCCCCAGGCCCAGCCUUGCCCACAAGAUUCCCAGAUUUC… | 11147 nt | 0.5070 |
| LINC01270:17 | AGACGGCCAUCCCCCAGGCCCAGCCUUGCCCACAAGAUUCCCAGAUUUC… | 9898 nt | 0.4941 |
| LINC01270:18 | CUUGCCCACAAGAUUCCCAGAUUUCACAACUGGGGCUGGCGCUGAAUCA… | 8516 nt | 0.4871 |
No relevant information is available at the moment.
A study in humans identified LINC01270 as a long non-coding RNA that is downregulated in myocardial fibrosis versus acute myocardial infarction and upregulated in heart failure versus myocardial fibrosis, suggesting a mechanistic role in this disease progression [Wang et al. DOI:10.3389/fcvm.2021.664044].