| ID | Sequence | Length | GC content |
|---|---|---|---|
| LINC01270:4 | CGCAGCCCAGCCCCCAAGCCCCGCCAGCGGGAAGCACCAGUUAGCCCAG… | 2483 nt | 0.4631 |
| LINC01270:5 | CAGAACUGCAGUGAGGGCUGUGAGUGUGGGGUGUCUCAACUGCACCUGG… | 2672 nt | 0.4966 |
| LINC01270:6 | GGGCUUUCCCUAAUUUCAAUCUCCCACCAGUCCUAUCAGCUUUACUGGG… | 3260 nt | 0.5117 |
| LINC01270:7 | AGAGGUGGUUGGGUCCUGAGAGCUCUGCCCUUAUGAAUGGAUUAGUAUC… | 943 nt | 0.5408 |
| LINC01270:8 | GGCAGACGUGUGAUGAAACACGUUUAUUACAGGAAGCACCAGUUAGCCC… | 510 nt | 0.5451 |
| LINC01270:9 | AUUAAUGACCCGGAGCCACAUGACGGAGCAUGACACAGCACUUUUGGGC… | 1133 nt | 0.5675 |
No relevant information is available at the moment.
A study in humans identified LINC01270 as a long non-coding RNA that is downregulated in myocardial fibrosis versus acute myocardial infarction and upregulated in heart failure versus myocardial fibrosis, suggesting a mechanistic role in this disease progression [Wang et al. DOI:10.3389/fcvm.2021.664044].